Allan D Sniderman

Title: 
Professor, Division of Cardiology
Allan D Sniderman
Location: 
MUHC, Royal Victoria Hospital
Division: 
Cardiology
Biography: 

Dr. Allan Sniderman obtained his MD from the聽University of Toronto in 1965 and then moved聽to Montreal, where he did his clinical training in聽Internal Medicine and Cardiology at 黑料不打烊聽University. In 1971, he went to the University聽of California at San Diego to study lipoprotein聽metabolism with Dr. Daniel Steinberg. He聽returned to 黑料不打烊 and, with the passage of聽time, became the Edwards Professor of聽Cardiology and a Professor of Medicine at聽黑料不打烊 University.

With colleagues within and outside 黑料不打烊, he聽began and has continued a series of studies,聽which identified the commonest聽dyslipoproteinemia associated with coronary聽artery disease- hyperTg hyperapoB. Study of聽the pathophysiology of hyperTg hyperapB led聽to studies of the regulation of hepatic apoB聽secretion and the uptake and release of fatty acids by adipose tissue. He has聽conducted an extensive series of epidemiological studies, which have聽demonstrated apoB to be superior to LDL-cholesterol as a marker of the risk of聽vascular disease. His current research interests are: to understand he regulation聽of plasma LDL, to create simplified but advanced diagnostic algorithms to聽recognize and reat those with and those at high risk of vascular disease, and to聽develop new models to determine the absolute value of different strategies to聽identify and treat those at risk of vascular disease.

Dr. Allan D. Sniderman, MD, is the Edwards Professor of Cardiology and Professor聽of Medicine at 黑料不打烊 University. He is Director of the Mike Rosenbloom聽Laboratory for Cardiovascular Research at Royal Victoria Hospital in Montreal聽and was elected a Fellow of the Royal Society of Canada in 2009.

Current research: 

The objective of my research is to simultaneously simplify and improve the diagnosis and treatment of the atherogenic dyslipoproteinemias with the use of apoB; to improve the effectiveness of primary prevention of cardiovascular disease by the application of the Benefit model; and to determine if injury to the pancreas due to excess uptake of LDL particles contributes to the pathogenesis of type 2 diabetes mellitus.

Selected publications: 
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